Geldanamycin

製品コードS2713 別名:NSC 122750

Geldanamycin化学構造

分子量(MW):560.64

Geldanamycin is a natural existing HSP90 inhibitor with Kd of 1.2 μM, specifically disrupts glucocorticoid receptor (GR)/HSP association.

サイズ 価格(税別)  
JPY 28220.00
JPY 44820.00
JPY 111220.00

カスタマーフィードバック(3)

  • Phenotypic effect of Genetic or Pharmacologic Compromise of the 477 Hsp70-StiA-Hsp90 Complex. The impact of each genetic modification on radial growth, conidiation, and response to various stress conditions was assessed after inoculation of a suspension of 104 conidia on glucose minimal medium (GMM) agar plates and incubation at 37ºC for 5 days.

    Antimicrob Agents Chemother, 2015, 10.1128/AAC.00946-15. Geldanamycin purchased from Selleck.

    C2C12 myoblasts were transfected with HA-tagged A17-PABPN1 constructs. Twenty-four hours post-transfection, cells were treated with CHX (10 μg/ml) alone or together with geldanamycin (2.5 μM) for the indicated times at 37°C. Lysates were blotted to show the expression of the proteins of interest. Band density was quantified and is shown in the line graph (right panels). Data are shown as the mean ± SEM (n = 5); **, P < 0.01.

    PLoS One, 2015, 10(9):e0138936.. Geldanamycin purchased from Selleck.

  • RT-qPCR analysis of eNOS mRNA and representative Western blot images of eNOS expression, respectively, in irradiated BAECs pretreated of geldanamycin (GA; 500 nM). At 12 h after 10 Gy irradiation, BAECs were harvested. The results suggest that both HSP90 is involved in the upregulation of eNOS in irradiated BAECs.

    Radiat Res, 2018, 189(5):519-528. Geldanamycin purchased from Selleck.

製品安全説明書

HSP (e.g. HSP90)阻害剤の選択性比較

生物活性

製品説明 Geldanamycin is a natural existing HSP90 inhibitor with Kd of 1.2 μM, specifically disrupts glucocorticoid receptor (GR)/HSP association.
ターゲット
p185 [4]
(SKBr3 cells)
HSP90 (N-terminal domain) [1]
(Cell-free assay)
HSP90 [1]
(Cell-free assay)
70 nM 0.78 μM(Kd) 1.2 μM(Kd)
体外試験

Geldanamycin binds in the ATP-binding site in the N-terminus domain of Hsp90s (residues 1-220). Geldanamycin inhibits the ATPase activity of Hsp90 in a dose-dependent manner. [1] Geldanamycin causes a dose-dependent G2 arrest and reversible inhibiton o f entry into the S phase in A2780 human ovarian cell line. This inhibition is accompanied by p53 increase and finally demonstrated to be p53 dependent. [2] Geldanamycin causes polyubiquitination and proteasomal degradation of the p185 receptor protein-tyrosin kinase and shows a IC50 with 70 nM. [3, 4] Geldanamycin is a typical anti-tumor reagent, shows a mean GI50 with 0.18 μM against the panel of 60 human tumor cell lines. [5]

細胞データ
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
A2780 cells NHOzNHpRem:uaX\ldoF1cW:wIHHzd4F6 NESzU5BEd22yb4Xu[EB4[XNiZY\hcJVifGWmIH\vdkBidnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIH;2ZZJq[W5iY3HyZ4lvd22jIHPlcIwhdGmwZTDBNlc5OCxiSVO1NF0{NjRizszN MVixNVUyPDF2NR?=
SW620 cell Mly5S5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? NUHuWYZtUW6qaXLpeI9zgSClb37j[Y51emG2aX;uJIFo[Wmwc4SgbJVu[W5iY3;sc5Jm[3SjbDDjZZJkcW6xbXGgV3c3OjBiY3XscEBtcW6nczygTWM2OD14LkKgcm0> MV2xOVY2QDh5OR?=
MCF-7 cell M4LxWGdzd3e2aDDpcohq[mm2aX;uJIF{e2G7 MUPJcohq[mm2b4L5JINwdmOnboTyZZRqd25iYXfhbY5{fCCqdX3hckBjemWjc4SgZ4Fv[2W{IF3DSk04KGOnbHygcIlv\XNuIFnDOVA:Pi53IH7N MYKxOVY2QDh5OR?=
SKBR3 cells MnfOVJJwdGmoZYLheIlwdiCjc4PhfS=> NH3Yc|M4OiCq NXnLPFJwSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBme3S{b3flckBz\WOncITvdkBl\W[rY3nlcpQhcHWvYX6gV2tDWjNiY3XscJMh[W[2ZYKgO|IhcHK|LDDJR|UxRThwNTDuUS=> MmfVNlM3PDhzOEC=
MCF7 cells NXP0[YdkWHKxbHnm[ZJifGmxbjDhd5NigQ>? Mly3O|IhcA>? M3mx[2FvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iTVPGO{Bk\WyuczDlfJBz\XO|aX7nJIV{fHKxZ3XuJJJm[2WydH;yJIFnfGW{IEeyJIhzeyxiSVO1NF06Njhibl2= NFjFVGkzOzZ2OEG4NC=>
MDA-kb2 cells NEDYUlFHfW6ldHnvckBie3OjeR?= MofvNVghcA>? NFPNTIRKdmirYnn0bY9vKG:oIFjTVFkxKGmwIHj1cYFvKE2GQT3rZlIh[2WubIOgZZN{\XO|ZXSgZZMhemWmdXP0bY9vKGmwIHfseYNw[2:{dHnjc4llKHKnY3XweI9zNWSncHXu[IVvfCCudXPp[oVz[XOnIHX4dJJme3Orb36gZYZ1\XJiMUigbJJ{KGK7IH\pdoVndHlibIXjbYZmemG|ZTDy[ZBwenSncjDn[Y5mKGG|c3H5MEBKSzVyPUGwJI5O MWqyOFk5PDl|Nh?=
human SK-BR-3 cells MoTEVJJwdGmoZYLheIlwdiCjc4PhfS=> NUD4OmFXSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDTT{1DWi1|IHPlcIx{NCCLQ{WwQVE2Njhibl2= NUPO[G97OTl6OU[4OFg>
HUVEC cells NWewfW9mS3m2b4TvfIlkyqCjc4PhfS=> NWS3Opc2PzJiaB?= MnzKR5l1d3SxeHnjbZR6KGGpYXnud5QhUFWYRVOgZ4VtdHNiYX\0[ZIhPzJiaILzJIJ6KE2WVDDhd5NigSxiSVO1NF0yQSCwTR?= NIXDfZMzPTJ5N{C2Oy=>
human HCT116 cells M4LSUWdzd3e2aDDpcohq[mm2aX;uJIF{e2G7 MXPHdo94fGhiaX7obYJqfGmxbjDv[kBpfW2jbjDIR3QyOTZiY3XscJMtKEeLNUC9NlEhdk1? NHzNTHcyQDJ2M{ewNy=>
K562 cell MnjyS5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? NUjqZlhwUW6qaXLpeI9zgSClb37j[Y51emG2aX;uJIFo[Wmwc4SgbJVu[W5ibHX1b4VucWFiS{W2NkBk\WyuIHzpcoV{NCCLQ{WwQVIzNjFibl2= MVWxOVY2QDh5OR?=
HT-29 cell NHjUdHpIem:5dHigbY5pcWKrdHnvckBie3OjeR?= NHnaSm9KdmirYnn0c5J6KGOxbnPlcpRz[XSrb36gZYdicW6|dDDoeY1idiClb3zvdoVkfGGuIHPhdoNqdm:vYTDIWE0zQSClZXzsJIxqdmW|LDDJR|UxRTJ2LkWgcm0> MknTNVU3PTh6N{m=
HCT116 cells NHnuNJRRem:uaX\ldoF1cW:wIHHzd4F6 M1HqOmFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iSFPUNVE3KGOnbHzzJIJ6KGy3bXnu[ZNk\W6lZTDhd5NigSxiRVO1NF0xNjB|IN88US=> NYHISHJNOjF4MEW5O|U>
NCI-H1975 cells MULQdo9tcW[ncnH0bY9vKGG|c3H5 M2O1UmFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iTlPJMWgyQTd3IHPlcIx{NCCLQ{WwQVM3KG6P NVmycnRqOjF5MUWxOlU>
human DLD1 cells Ml;RS5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? M{fhNGdzd3e2aDDpcohq[mm2aX;uJI9nKGi3bXHuJGRNTDFiY3XscJMtKEeLNUC9N|chdk1? NIj4O|EyQDJ2M{ewNy=>
human A431 cells MWLDfZRwfG:6aXRCpIF{e2G7 NWXzVpBwPzJiaB?= Mke3R5l1d3SxeHnjbZR6KGGpYXnud5QhcHWvYX6gRVQ{OSClZXzsd{Bi\nSncjC3NkBpenNiYomgUXRVKGG|c3H5MEBKSzVyPUSwJI5O MWGyOVI4PzB4Nx?=
human HepG2 cells NWXtNWxQS3m2b4TvfIlkyqCjc4PhfS=> MUe3NkBp NVPpfXAzS3m2b4TvfIlkcXS7IHHnZYlve3RiaIXtZY4hUGWyR{KgZ4VtdHNiYX\0[ZIhPzJiaILzJIJ6KE2WVDDhd5NigSxiSVO1NF01OCCwTR?= NHfnPG0zPTJ5N{C2Oy=>
human BGC823 cells MlTjR5l1d3SxeHnjxsBie3OjeR?= MnSxO|IhcA>? M4LJb2N6fG:2b4jpZ4l1gSCjZ3HpcpN1KGi3bXHuJGJISzh{MzDj[YxteyCjZoTldkA4OiCqcoOgZpkhVVSWIHHzd4F6NCCLQ{WwQVQxKG6P M{\h[lI2Ojd5ME[3
human SKBR3 cells NX:0RXNCS3m2b4TvfIlkyqCjc4PhfS=> NET3e5Y4OiCq Mn30R5l1d3SxeHnjbZR6KGGpYXnud5QhcHWvYX6gV2tDWjNiY3XscJMh[W[2ZYKgO|IhcHK|IHL5JINmdGy2aYTldk1odG9iYYPzZZktKEmFNUC9OFEhdk1? NUnFcldROTl2MEW1Nlg>
human MDA-MB-231 cells NWTBeGw6S3m2b4TvfIlkyqCjc4PhfS=> NXjGS|NmPzJiaB?= MmnTR5l1d3SxeHnjbZR6KGGpYXnud5QhcHWvYX6gUWRCNU2ELUKzNUBk\WyuczDh[pRmeiB5MjDodpMh[nliTWTUJIF{e2G7LDDJR|UxRTVyIH7N MVeyOVI4PzB4Nx?=
human A549 cells MXjHdo94fGhiaX7obYJqfGmxbjDhd5NigQ>? NF\OTlZIem:5dHigbY5pcWKrdHnvckBw\iCqdX3hckBCPTR7IHPlcIx{NCCJSUWwQVY1KG6P M4HKSVE5OjR|N{Cz
Sf9 cells NX;JU|MxTnWwY4Tpc44h[XO|YYm= M1;HemRqe3CuYXPlcYVvfCCxZjDHUU1DV0SLUGmg[pJwdSCqdX3hckBnfWyuIHzlcod1cCCKU2C5NEBidHCqYTDlfJBz\XO|ZXSgbY4h[mGldXzveolzfXNvaX7m[YN1\WRiU3[5JINmdGy|IHHmeIVzKDF4IHjyd{BjgSCobIXvdoV{[2WwY3WgdI9t[XKrenH0bY9vKGG|c3H5MEBKSzVyPUe0JI5O MkPmNlQ4PTF2NEG=
human U87MG cells NEnTSXVRem:uaX\ldoF1cW:wIHHzd4F6 NET2[Y9CdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIGW4O21IKGOnbHzzMEBKSzVyPUi5JI5O NGP6PYUzOTdzNUG2OS=>
human A549 cells MXrDfZRwfG:6aXRCpIF{e2G7 NFW1cWM4OiCq NEPIOpNEgXSxdH;4bYNqfHliYXfhbY5{fCCqdX3hckBCPTR7IHPlcIx{KGGodHXyJFczKGi{czDifUBOXFRiYYPzZZktKEmFNUC9PVchdk1? NFvwVJAzPTJ5N{C2Oy=>
mouse P19 cells NWHoOnhKS3m2b4TvfIlkyqCjc4PhfS=> M1TpfFE5KGh? NVf0eIlzS3m2b4TvfIlkcXS7IHHnZYlve3RibX;1d4UhWDF7IHPlcIx{KGGodHXyJFE5KGi{czygTWM2OD1yLkGg{txO NXO3VJZWOTd2NEK1OlU>
human HL7702 cells NHHCO2FEgXSxdH;4bYPDqGG|c3H5 NWXwZmlWPzJiaB?= M1XUXGN6fG:2b4jpZ4l1gSCjZ3HpcpN1KGi3bXHuJGhNPzdyMjDj[YxteyCjZoTldkA4OiCqcoOgZpkhVVSWIHHzd4F6NCCLQ{WwQVAvOTRzIN88US=> NVzsUIpPOjV{N{ewOlc>
human A549 cells NGqydXZEgXSxdH;4bYPDqGG|c3H5 MojsNkBl[Xm| MkjlR5l1d3SxeHnjbZR6KGGpYXnud5QhcHWvYX6gRVU1QSClZXzsd{Bi\nSncjCyJIRigXNiYomgRYxidWG{Qnz1[UBie3OjeTygTWM2OD1yLkG1JO69VQ>? NGj4ZnEzOzl2N{e5OC=>
human A431 cells M{TkWXBzd2yrZnXyZZRqd25iYYPzZZk> M1nmeVczKGh? M2P5S2FvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iQUSzNUBk\WyuczDh[pRmeiB5MjDodpMtKEmFNUC9NE4zKM7:TR?= NIjySFAzODZ3NUKzOy=>
human HepG2 cells MYPDfZRwfG:6aXRCpIF{e2G7 MULDfZRwfG:6aXPpeJkh[WejaX7zeEBpfW2jbjDI[ZBIOiClZXzsd{BjgSCPVGSgZZN{[XluIFnDOVA:OC5|IN88US=> NHnvWFUzOzZ3NkW1Oi=>
human SW480 cells NGD5emVEgXSxdH;4bYPDqGG|c3H5 MVu3NkBp MWfDfZRwfG:6aXPpeJkh[WejaX7zeEBpfW2jbjDTW|Q5OCClZXzsd{Bi\nSncjC3NkBpenNiYomgUXRVKGG|c3H5MEBKSzVyPUCuN|Eh|ryP NVLicplbOjV{N{ewOlc>
human LNCAP cells Mn3sR5l1d3SxeHnjxsBie3OjeR?= NYf5PIhKPzJiaB?= MoS2R5l1d3SxeHnjbZR6KGGpYXnud5QhcHWvYX6gUG5ESVBiY3XscJMh[W[2ZYKgO|IhcHK|IHL5JG1VXCCjc4PhfUwhUUN3ME2wMlQ{KM7:TR?= M2T2ZVI2OTB3OUK0
human LS174T cells NUe5b3NKS3m2b4TvfIlkyqCjc4PhfS=> M1LROmN6fG:2b4jpZ4l1gSCjZ3HpcpN1KGi3bXHuJGxUOTd2VDDj[YxteyCkeTDNWHMh[XO|YYmsJGlEPTB;MD60OUDPxE1? NYrXd5VTOTdyM{SxN|U>
human HeLa cells MXzDfZRwfG:6aXRCpIF{e2G7 NIPjRXM4OiCq MmXZR5l1d3SxeHnjbZR6KGGpYXnud5QhcHWvYX6gTIVN[SClZXzsd{Bi\nSncjC3NkBpenNiYomgUXRVKGG|c3H5MEBKSzVyPUCuO|k5KM7:TR?= MWOyOVI4PzB4Nx?=
rat L6 cells NUCzNIRKS3m2b4TvfIlkyqCjc4PhfS=> MmrQO|IhcA>? M{nUTWN6fG:2b4jpZ4l1gSCjZ3HpcpN1KHKjdDDMOkBk\WyuczDh[pRmeiB5MjDodpMh[nliQXzhcYFzKEKudXWgZZN{[XluIFnDOVA:PSEQvF2= Mm\qNlQ2QDB3M{G=
human MCF7 cells MX3DfZRwfG:6aXRCpIF{e2G7 NEDaVZlEgXSxdH;4bYNqfHliYXfhbY5{fCCqdX3hckBOS0Z5IHPlcIx{KGK7IGPSRkBie3OjeTygTWM2OD17Lk[g{txO NXfZS3N1OTl3NkCzOVM>
human MCF7 cells MXHHdo94fGhiaX7obYJqfGmxbjDhd5NigQ>? NHvr[VVIem:5dHigbY5pcWKrdHnvckBw\iCqdX3hckBOS0Z5IHPlcIx{KGGodHXyJIRigXNiYomgV3JDKGG|c3H5MEBIUTVyPUO1MlYh|ryP MkeyNVc5PjlyOUi=
HEK293T cells NVfsWXZ5TnWwY4Tpc44h[XO|YYm= NHHuUJBKdmirYnn0bY9vKG:oIGTOSk1idHCqYT3pcoR2[2WmIF7GMYtieHCjQjDhZ5RqfmG2aX;uJIV5eHKnc4Pl[EBqdiCKRVuyPVNVKGOnbHzzJIJ6KGy3Y3nm[ZJie2VicnXwc5J1\XJiZ3Xu[UBie3OjeR?= MUKxPFQxQDdzMx?=
human Jurkat cells NWD2THRLTnWwY4Tpc44h[XO|YYm= MXvJcohq[mm2aX;uJI9nKFSQRj3hcJBp[S2rbnT1Z4VlKE6ILXvhdJBiSiCjY4TpeoF1cW:wIHX4dJJme3OnZDDpckBHSUSGIHTl[olkcWWwdDDoeY1idiCMdYLrZZQh[2WubIOgZpkhdHWlaX\ldoF{\SC{ZYDvdpRmeiCpZX7lJIF{e2G7 MmDjNVg1ODh5MUO=
human SKBR3 cells MULGeY5kfGmxbjDhd5NigQ>? NHHU[Y0zPCCq M3nlTmlvcGmkaYTpc44hd2ZiSIPwPVAudWWmaXH0[YQhUEWUMjDk[Ydz[WSjdHnvckBqdiCqdX3hckBUU0KUMzDj[YxteyCjZoTldkAzPCCqcoOgZpkhX2W|dHXyckBjdG:2 MnvjNVg5OTZzMUG=

他の多くの細胞株試験データをご覧になる場合はこちらをクリックして下さい

体内試験 Geldanamycin (50 mg//kg) shows 30% inhibition on pl85-associated phosphotyrosine levels in FRE/erbB-2 mice. [6]

お薦めの試験操作(参考用のみ)

キナーゼ試験:

[1]

+ 展開

Isothermal Titration Calorimetry (ITC) of Nucelotide Binding:

The titration experiments are performed using the MSC system. In each experiment, 16 aliquots of 15 μL of geldanamycin (300 μM in 1% DMSO) are injected into 1.3 mL of protein (31 μM in 20 mMTris-HCl, pH 7.5, 1 mMEDTA) at 25 °C, and the resulting data are fit after subtracting the heats of dilution. Heats of dilution are determined in separate experiments from addition of geldanamycin into buffer and buffer into protein. No evidence for binding of DMSO in the nucleotide binding site is observed. Titration data are fit using a nonlinear least-squares curve-fitting algorithm with three floating variables: stoichiometry, binding constant (Kb) 1/Kd), and change of enthalpy of interaction (ΔH°). Dissociation constants estimated for geldanamycin binding to intact yeast Hsp90 is 1.22 μM, and for binding to Hsp90 N-terminal domain is 0.78 μM. No meaningful heat is observed with binding to the C-terminal fragment.
細胞試験:

[2]

+ 展開
  • 細胞株: A2780 human ovarian cell line
  • 濃度: 0.001-10 μM
  • 反応時間: 3 hours
  • 実験の流れ:

    Exponentially growing cells are treated with Geldanamycin and at various times DNA synthesis is assessed by incorporation of bromodeoxyuridine (BrdUrd) and flow cytometric analysis. No marked difference in total cell number is noted during this time course for treated and untreated cultures. BrdUrd (10 μM) is incorporated over a 4-h incubation period at 37 °C and cells are harvested and fixed in 70% ethanol. After denaturation of the DNA with 2 N HC1, cells are incubated with an anti-BrdUrd mouse monoclonal antibody followed by a fluorescein isothiocyanate (FITC)-linked goat anti-mouse IgG. Cells are stained for 30 minutes at room temperature with propidium iodide and analysed by flow cytometry using a Coulter EPICS Profile Analyzer.


    (参考用のみ)
動物試験:

[6]

+ 展開
  • 動物モデル: FRE/erbB-2 tumors in nu/nu mice
  • 製剤: Geldanamycin is dissolved in DMSO.
  • 投薬量: 50 mg/kg
  • 投与方法: Administered via i.p.
    (参考用のみ)

溶解度 (25°C)

体外 DMSO 36 mg/mL (64.21 mM) warming
Water Insoluble
Ethanol Insoluble

* 溶解度測定はSelleck技術部門によって行われており、その他文献に示されている溶解度と差異がある可能性がありますが、同一ロットの生産工程で起きる正常な現象ですからご安心ください。

化学情報

分子量 560.64
化学式

C29H40N2O9

CAS No. 30562-34-6
保管
in solvent
別名 NSC 122750

便利ツール

モル濃度計算器

モル濃度計算器

求めたい質量、体積または濃度を計算してください。

質量 (g) = 濃度 (mol/L) x 体積 (L) x 分子量 (g/mol)

モル濃度計算器方程式

  • 質量
    濃度
    体積
    分子量

*貯蔵液を準備するとき、常に、オンであるとわかる製品のバッチに特有の分子量を使って、を通してラベルとMSDS/COA(製品ページで利用可能な)。

希釈計算器

希釈計算器

貯蔵液を準備するために必要な希釈率を計算してください。Selleck希釈計算器は、以下の方程式に基づきます:

開始濃度 x 開始体積 = 最終濃度 x 最終体積

希釈の計算式

この方程式は、一般に略語を使われます:C1V1 = C2V2 ( 入力 出力 )

  • C1
    V1
    C2
    V2

常に貯蔵液を準備するとき、小びんラベルとMSDS/COA(オンラインで利用できる)で見つかる製品のバッチに特有の分子量を使ってください。

連続希釈計算器方程式

  • 連続希釈剤

  • 計算結果

  • C1=C0/X C1: LOG(C1):
    C2=C1/X C2: LOG(C2):
    C3=C2/X C3: LOG(C3):
    C4=C3/X C4: LOG(C4):
    C5=C4/X C5: LOG(C5):
    C6=C5/X C6: LOG(C6):
    C7=C6/X C7: LOG(C7):
    C8=C7/X C8: LOG(C8):
分子量計算器

分子量计算器

そのモル質量と元素組成を計算するために、合成物の化学式を入力してください:

総分子量:g/mol

チップス: 化学式は大文字と小文字の区別ができます。C10H16N2O2 c10h16n2o2

モル濃度計算器

質量 濃度 体積 分子量

技術サポート

ストックの作り方、阻害剤の保管方法、細胞実験や動物実験の際に注意すべき点など、製品を取扱う時に問い合わせが多かった質問に対しては取扱説明書でお答えしています。

Handling Instructions

他に質問がある場合は、お気軽にお問い合わせください。

  • * 必須

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HSP (e.g. HSP90) Inhibitors with Unique Features

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Tags: Geldanamycinを買う | Geldanamycin ic50 | Geldanamycin供給者 | Geldanamycinを購入する | Geldanamycin費用 | Geldanamycin生産者 | オーダーGeldanamycin | Geldanamycin化学構造 | Geldanamycin分子量 | Geldanamycin代理店
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大阪市内で「G20サミット」が開催されることに伴い、大阪府内で大規模な交通規制が行われる影響で、6月26日(水)から7月1日(月)までの間、下記地域へ出荷が行えなくなります。
対象地域
大阪府(全域)、兵庫県(芦屋市、尼崎市、伊丹市、西宮市)
上記地域より西の地域につきましては納期の遅延が予想されます。
また、国際便も遅延が予想されているため国内在庫がない製品の納期が一週間程度遅れる可能性がございます。
ご不便をおかけいたしますが、宜しくお願い申し上げます。

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細胞株 試験類型 濃度 培養時間 溶剤類型 活性叙述 PMID